Val(8)GLP-1 rescues synaptic plasticity and reduces dense core plaques in APP/PS1 mice

Keyphrases

• Synaptic Plasticity 100%
• Plaque 100%
• Dense Cores 100%
• Glucagon-like peptide-1 (GLP-1) 100%
• Amyloid Precursor Protein 100%
• Long-term Potentiation 42%
• Diabetes 28%
• Amyloid Plaques 28%
• Alzheimer's Disease Model 28%
• Synaptic 14%
• Intraperitoneal Injection 14%
• Risk Factors 14%
• Blood-brain Barrier 14%
• Aged Mice 14%
• Alzheimer's Disease 14%
• Cortex 14%
• Glucagon-like 14%
• Incretin Hormones 14%
• Age-related 14%
• Inflammation Response 14%
• Congo Red 14%
• Neurodegenerative Processes 14%
• Wild Mice 14%
• Paired-pulse Facilitation 14%
• Presenilin 14%
• Microglial Activation 14%
• Response Formation 14%
• Degenerative Process 14%
• Plaque Formation 14%

Biochemistry, Genetics and Molecular Biology

• Mouse 100%
• Long Term Potentiation 100%
• Amyloid Precursor Protein 100%
• Nerve Cell Plasticity 100%
• Age 66%
• Enzyme 33%
• Glucagon-Like Peptide-1 33%
• Incretin 33%
• Amyloid 33%
• Congo Red 33%
• Mouse Model 33%
• Presenilin 33%
• Blood Brain Barrier 33%
• Amyloid Beta 33%
• Microglia 33%
• Facilitation 33%
• Wild Type Mouse 33%

Medicine and Dentistry

• Glucagon Like Peptide 1 100%
• Plaque 100%
• Amyloid Precursor Protein 100%
• Alzheimer's Disease 37%
• Long-Term Potentiation 37%
• Age 25%
• Diabetes 25%
• Amyloid Plaque 25%
• Therapeutic Procedure 12%
• Intraperitoneal Drug Administration 12%
• Serositis 12%
• Enzyme 12%
• Incretin 12%
• Congo Red 12%
• Microglia 12%
• Synapsin I 12%
• Blood Brain Barrier 12%
• Facilitation 12%
• Presenilin 1 12%